H3K27me3 conditions chemotolerance in triple-negative breast

H3K27me3 conditions chemotolerance in triple-negative breast

A benchmark of computational pipelines for single-cell histone

An EZH2-NF-κB regulatory axis drives expression of pro-oncogenic

The loss of B7-H4 expression in breast cancer cells escaping from T cell cytotoxicity contributes to epithelial-to-mesenchymal transition, Breast Cancer Research

Cancers, Free Full-Text

Simulating cell-free chromatin using preclinical models for cancer

Quantitative normalization resolves distinct chromatin landscapes

Molecular and functional profiling of chemotolerant cells unveils

H3K27me3 conditions chemotolerance in triple-negative breast

The loss of B7-H4 expression in breast cancer cells escaping from T cell cytotoxicity contributes to epithelial-to-mesenchymal transition, Breast Cancer Research

Estrogen-induced reactive oxygen species, through epigenetic

H3K27me3 conditions chemotolerance in triple-negative breast